【摘要】 目的 調查汶川地震傷殘人員的日常生活活動能力(ADL),分析影響傷殘人員日常生活活動能力的相關因素,為汶川地震傷殘人員的社區康復提供依據。方法 利用ADL量表對283名汶川地震傷殘人員的日常生活活動能力進行測量。結果 283名傷殘人員地震傷殘人員ADL平均得分為85.08±14.73,其中20.8%ADL完全正常,71.0%的傷殘人員ADL日常生活能力輕度障礙,8.2%ADL中重度障礙；經多因素分析,地震傷殘人員ADL的主要影響因素依次是:傷殘程度、傷殘合并疾病、傷殘并發癥、是否參與殘疾人組織或相關活動(Plt;0.05)。結論 傷殘人員ADL關鍵在于殘疾程度,避免傷殘并發癥和二次殘疾的發生,控制傷殘合并疾病的發生,增加傷殘人員的社會參與度,均有利于傷殘人員ADL的提高。
Adenoid hypertrophy in children with epilepsy is rarely reported. This paper analyzes the clinical characteristics and incidence of adenoid hypertrophy in children with epilepsy.Methods The clinical data in children with epilepsy from December 2014 to April 2020 in Shenzhen Children's hospital were analyzed retrospectively.Results There were 449 cases diagnosed with adenoid hypertrophy (2.74%) in 16387 children with epilepsy. Among 449 cases of adenoid hypertrophy, 276 males (61.47%) and 173 females (38.53%). The age distribution was: 28 days to 1 year old, 8 cases (2%); 1-3 years old, 78 cases (17%); 3-6 years old 167 cases (37%); 6-12 years old, 153 cases (34%); 12-18 years old, 43 cases (10%). In 40 patients the IgG antibody were positive for EB capsid antigen in 25 (62.5%). In 56 cases of EB virus DNA were detected by fluorescence quantitative PCR, 25 (44.64%) positive, and 21/44 cases (47.72%) were positive by general nucleic acid detection of enteroviruses. The neutrophil reduction rate in peripheral blood was 42.19% in 673 tests, lymphocyteincreased in 292 (43.38%), platelet count increased in 307 (45.61%), abnormal in platelet hematocrit in 311 (46.21%); the mean volume of RBC was decreased in319 (47.39%) tests. The content of \begin{document}${\rm{HCO}_3^-} $\end{document} was reduced in 20/55 cases (36.36%). 25-hydroxy vitamin D was 33 (44.5%) decreased in 74 cases. The blood glucose was measured in 146 cases, 60 (41.09%) increased, total cholesterol was 31 (40.78%) increased in 76 cases, serum C peptide was 12 (29.26%) increased in 41 cases.Conclusion Adenoid hypertrophy in children with epilepsy may be related to infection, inflammation or immune disorder, which may cause nutritional, metabolic or internal environment disorders. Therefore, there is need of nursing and health education, transferring to specialized centers for diagnosis and treatment.